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Swine flu's symptoms in human are similar to the symptoms of regular human flu, that include fever, cough, sore throat, body aches, headache, chills and fatigue. Some people have reported diarrhea and vomiting with swine flu. In the past, severe illness (pneumonia and respiratory failure) and deaths have been reported with swine flu infection in people. Like, seasonal flu, swine flu may cause a worsening of underlying chronic medical conditions.

Spread of this swine influenza A (H1N1) virus is thought to be happening in the same way that seasonal flu spreads. Flu viruses are spread mainly from person to person through coughing or sneezing of people with influenza. Sometimes people may become infected by touching something with flu viruses on it and then touching their mouth or nose. Infected people may be able to infect others beginning 1 day before symptoms develop and up to 7 or more days after becoming sick. That means that we may be able to pass on the flu to someone else before you know we are sick, as well as while we are sick.

To prevent of infection always remember to wash your hands. Try to stay in good general health. Get plenty of sleep, be physically active, manage your stress, drink plenty of fluids, and eat nutritious food. Try not touch surfaces that may be contaminated with the flu virus. Avoid close contact with people who are sick. Some viruses and bacteria can live 2 hours or longer on surfaces like cafeteria tables, doorknobs, and desks. Frequent hand washing will help you reduce the chance of getting contamination from these common surfaces.

Germs can be spread when a person touches something that is contaminated with germs and then touches his or her eyes, nose, or mouth. Droplets from a cough or sneeze of an infected person move through the air. Germs can be spread when a person touches respiratory droplets from another person on a surface like a desk and then touches their own eyes, mouth or nose before washing their hands.

CDC recommends the use of oseltamivir or zanamivir for the treatment and/or prevention of infection with these swine influenza viruses. Antiviral drugs are prescription medicines (pills, liquid or an inhaler) that fight against the flu by keeping flu viruses from reproducing in your body. If you get sick, antiviral drugs can make your illness milder and make you feel better faster. They may also prevent serious flu complications. For treatment, antiviral drugs work best if started soon after getting sick (within 2 days of symptoms).

People with swine influenza virus infection should be considered potentially contagious as long as they are symptomatic and possible for up to 7 days following illness onset. Children, especially younger children, might potentially be contagious for longer periods.

"In large emergencies, such as those caused by earthquakes, or floods, some countries have lost as much as 50 percent of their hospital capacity to respond right at the time when life-saving functions and services were most acutely needed," Chan told a news conference in Beijing.

"When a new hospital is built, construction specifically designed to withstand earthquakes and extreme weather events adds about four percent to the cost," she said. "This is a very small amount when you consider the huge investment in a hospital, and the huge investment lost when a hospital collapses."

The WHO pointed to disasters such as the 2004 Indian Ocean tsunami in which around two-thirds of health facilities in Indonesia's badly hit Aceh province were damaged. Pakistan and Algeria suffered similarly during recent earthquakes.

Hospitals were also damaged during last year's massive earthquake in southwest China's Sichuan province, in which approximately 80,000 people died.

"Experience in Latin America and the Caribbean shows that retrofitting a structurally sound facility will cost no more than one percent of the hospital's budget," Chan said.

"Abundant experience demonstrates the tremendous pay-off... when hospitals remain standing and functioning as beacons of security and solidarity in the midst of disaster and despair."

For these reasons it is important that you come up with a plan of attack in order to quit smoking: although the cold-turkey technique works for some people, the vast majority of smokers will have success only with a more comprehensive plan. When you first consider the prospect of quitting smoking, it's probably going to seem far fetched, but keep in mind that thousands of people - people that are no different from you - quit smoking every year. If they can do it, there's no reason that you can't. Many smokers also feel that after a certain age it is "too-late" to quit smoking. Simply put, this isn't true, and should not be used as an excuse to avoid an attempt to quit smoking: the health benefits of quitting smoking begin the very day you stop.

Before you actually have your last cigarette, begin to build up your willpower. Your willpower is going to be your most important tool in quitting, and it's very unlikely that you will be successful without it. Spend some time thinking of the reasons you want to quit smoking. Learn about the health benefits of quitting, for both yourself and the people around you. Do some math and come up with some figures for the amount of money you'll save by not buying cigarettes, and think of something you'll use that money for.

Once you've built up your willpower, it's time to have your last cigarette. To keep your spirits up, understand that the human body is incredibly resilient, and your health will improve as soon as you stop smoking - literally. 8 hours after your last cigarette, carbon monoxide levels and oxygen levels in your blood stream will return to normal. At 24 hours after your last cigarette, you statistically reduce your chance of a heart attack. Only 48 hours after your last cigarette, your sense of taste and smell will improve as your nerve endings start growing. As you continue to stay smoke free, think of the longer-term benefits to quitting in order to keep your willpower up: even after 2 weeks your lung power will begin to increase, and continue to do so over time.

Other aspects of your health will continue to improve in various ways. The ultimate motivator should be the knowledge that 15 years after quitting, your risk of death is almost the same as someone who has never smoked - a remarkable fact that illustrates our the human body's surprising ability to restore itself. By coming up with a concrete plan to quit smoking you will greatly increase your chances of success. Crucial is understanding the important role that your willpower will play in the process, and planning to build up this willpower weeks before you attempt to quit. Once you've stopped you have to keep the strength of this willpower up, and to do so, remind yourself of the health benefits you will be privy to immediately after butting out that last cigarette.

Millions of men and women are now obsessed about how many calories they burn per workout, because of these inventions. You're probably one of these people, watching every calories as it creeps up ever so often during a slow cardio workout. Without taking to mind that you can wipe out a thirty-minute, 300-calorie cardio session with one mouthful of a glazed donut. There are far too many people being mislead to believe that if they don't burn 300-500 calories per workout, then they will not lose weight. After all, isn't this what you've been told over and over again in those shinny stylish fashion/fitness magazines?

There are many problems with this kind of approach to weight loss. For the record, it's hard to tell if the calorie counter machines are even accurate enough. One story on CBS news proved that calorie counting machines overestimated calorie burning numbers up to twenty percent!

If you are still depending on slow cardio for progressive fat loss, you will find that it is relatively useless and at the very least, ineffective. It will take a long time for you to burn a large number of calories and one study has shown that men who only used cardio workouts for fat loss ended up with a lower resting metabolic rate. By only depending on cardio you will basically undo the calorie burning you are trying to achieve. On the other hand, men in the same study that used weight training did not undergo a lower metabolism.

So what is the recommendation to losing fat in a faster and more effective way? The answer is to use weight lifting and interval workouts to burn less calories in less exercise time, but with a high intense form of training.

Your body will burn greater amounts of calories after exercise using intervals than it would after doing slow cardio and your metabolic rate will stay high. A fair amount of experts call this method the "after burn effect". So how are you supposed to do intervals, you may ask? Well, you could do a sprint for thirty seconds and rest for ninety seconds and redo that for six sets - using the bicycle is preferred or the treadmill if you are familiar with it.

Within that short time span the interval training will cause your muscles to go wild with energy. I like to call it a metabolic turbulence. This insane metabolic boost causes lots of calories to burn after exercise to get your body back under control. The results will cause you to burn more calories and fat in the post exercise period as your body attempts to get things back to normal.

Now there is a single time that you would want to count your calories, but that is only when you are counting up to determining how many calories you take in per day. Remember, you can wipe out an entire calorie burning workout in less than one minute just by eating junk food. Without some discipline and structure in your nutrition, there is nothing that any program can do to help you lose weight. So there you have it, workout nutrition discipline and interval training. These are the two anti-calorie counting methods that will help you get lean by losing fat.

About the Author

James S. Wallace http://www.yourhealthevolution.com/

Having identified 10 common variants of genes that modify the timing of the contraction of the heart, known as the QT interval, scientists in the Johns Hopkins University School of Medicine, in collaboration with an international contingent of researchers, now provide new insight about the underpinnings of the QT interval which, when prolonged or shortened, predisposes to sudden cardiac death.

QT interval, which is determined from a standard electrocardiogram (ECG), reflects the time it takes for the heart (ventricles) to contract and then reset for the next heartbeat.

Publishing March 22 in Nature Genetics, the international team including researchers from the Technical University in Munich, Johns Hopkins and others, used DNA samples previously collected for epidemiological studies to analyze the genomes of 15,842 individuals whose QT intervals had been measured by electrocardiogram. With DNA microarray chips, each able to assess hundreds of thousands of markers in each sample, followed by bioinformatic techniques to increase the number of markers, the researchers screened approximately 2.5 million markers to detect subtle alterations in the sequences of these genomes that modify the QT interval.

By focusing on 2.5 million sites in a genome of 3 billion sites, the scientists surveyed one-one-thousandth of nearly 16,000 genomes. This relatively small but still extremely powerful screen correlates genomic architecture with QT intervals, according to Aravinda Chakravarti, Ph.D., a professor in the McKusick-Nathans Institute of Genetic Medicine.

These common variants at 10 locations across the genome represent perhaps dozens of yet-to-be-identified genes that affect this trait, Chakravarti adds. Of the 10, one that had been previously identified — Nos1ap — was confirmed. Several others were suspected culprits, the effects of which hadn't been demonstrated in preliminary screens.

However, almost half were surprising new genes that no one would have guessed as being involved in cardiac biology, says Dan Arking, Ph.D., an assistant professor in the McKusick-Nathans Institute of Genetic Medicine. So it really does open up a new world of investigation because these are genes that would have never come up if we had only focused on a list of known candidate genes.

A separate study, led by Christopher Newton-Cheh, M.D., M.P.H., of the Massachusetts General Hospital Center for Human Genetic Research and Cardiovascular Research Center , found similar results from more than 13,000 individuals. We were very reassured to see such strong replication in two independent studies, says Newton-Cheh.

While any single genetic variation in any one individual does not necessarily imply a significant alteration to QT interval, much less increased risk of sudden cardiac death, there is meaning that resides in the collective.

The power of this genetic analysis is a result of screening many thousands of samples, says Chakravarti: We're not very good at predicting what happens to any one, single sample. It's sort of like, I could examine in great detail how important my vote was in the last election, but it's trivial compared to the collective vote. An individual's genome is important as part of the study's whole, but individually, it's of little consequence.

Likewise, if scientists analyze the effect on QT interval by any one of the genetic variants, the alteration amounts to just a couple milliseconds, which is not a huge amount, says Arking: But if you put all 10 genetic variants together, that bumps up the QT interval by about 20 milliseconds, which is significant.

This latest study builds on research published in 2006, when a screen of 100,000 sites in individuals of European ancestry first showed that the Nos1ap gene is associated with the QT interval; and subsequent research showing that sequence changes in Nos1ap are also a risk factor for sudden cardiac death. A third paper, published in January 2009 in PLoS one, widened the original screen to include multiethnic populations; that study confirmed that Nos1ap genetic variants alter QT interval in all populations and, in fact, have a stronger effect in women than men.

The reason people die from this cardiovascular disorder is because we know nothing about the antecedents, Chakravarti says. It's like a truck barreling down a slope: there's no way to stop it. The only way out is to understand the science of this in a deep, meaningful way. If we know, we can begin to intervene.

The research was supported in part by National Heart, Lung and Blood Institute, National Human Genome Research Institute, National Institute on Aging, National Institutes of Health, Donald W. Reynolds Cardiovascular Clinical Research Center at Johns Hopkins University, German Federal Ministry of Education and Research, Fondation Leducq, State of Bavaria, Ministry of Health of the Autonomous Province of Bolzano, South Tyrolean Sparkasse Foundation, and the Heinz Nixdorf Foundation.

Authors of the paper, in addition to Chakravarti and Arking, are Georg B. Ehret, Anna Kottgen, W.H. Linda Kao, Josef Coresh and Man Li, Johns Hopkins University; Arne Pfeufer and Christine Happle, Technical University Munich, Germany; Serena Sanna, Gianluca Usala, Mariano Dei, Silvia Naitza and Marco Orru, Istituto di Neurogenetica e Neurofarmacologia, Cagliari, Italy; Martina Muller and H.-Erich Wichmann, Helmholtz Center Munich; Gerhard Steinbeck and Stefan Kaab, Klinikum Grosshadern, Munich; Goncalo R. Abecasis and Vesela Gateva, University of Michigan; Christian Fuchsberger, Peter P. Pramstaller and Andrew A. Hicks, EURAC European Academy, Italy; Siegfried Perz, Helmholtz Center Munich; Maja Barbalic and Eric Boerwinkle, University of Texas Health Science Center; Benno Putz and Bertram Muller-Myhsok, Max Planck Institute of Psychiatry, Munich; Angelo Scuteri, Istituto Ricovero e Cura per Anziani, Rome; Ronald J. Prineas, Wake Forest University School of Medicine; Samer S. Najjar and Edward Lakatta, National Institute on Aging; Thomas W. Muhleisen, University of Bonn; Stefan Mohlenkamp and Karl-Heinz Jockel, University Hospital of Essen, University Duisburg-Essen; and David Schlessinger, National Institute on Aging.

CONTACTS: Johns Hopkins Medicine Media Relations and Public Affairs: Maryalice Yakutchik, 443-287-2251, myakutc1@jhmi.edu or Audrey Huang, 410-614-5105, audrey@jhmi.edu

NOTE TO EDITORS: Video clips of Chakravarti and Arking are available at: http://www.hopkinsmedicine.org/Press%5freleases/2009/03%5f22%5f09. .html

ON THE WEB:

http://www.hopkinsmedicine.org/geneticmedicine/

http://www.nature.com/ng/index.html

Paramedic
Requirements:
Candidate must possess at least a Bachelor’s Degree in Nursing, Pharmacy/Pharmacology, Medicine or equivalent.
At least 3 year(s) of working experience in the related field is required for this position.
Male, 28-35 years old
Certified Emergency Response for Trauma (Basic Trauma Life Support)
Certified Emergency Response for Cardiac (Basic Cardiac Life Support)
Certified for First Aid & Basic Life Support
Indonesia Manpower Ministry Occupational Health Certificate (Hiperkes)
Able to communicate in English (oral & written)
Computer skill
Certified in Medical Fitness

Medical Personel (Doctor)
Requirements:

• Candidate must possess at least a Bachelor’s Degree in Medical Science
• Male, 28-40 Years Old
• At least 3 year(s) of working experience in Medical Emergency Response.
• Certified Emergency Response for Trauma (Advance Trauma Life Support/ATLS)
• Certified Indonesia Man Power Occupational Health (HIPERKES)
• Certified Advance Cardio Life Support/ACLS
• Able to communicate in English both oral and written
• Computer Skill
• Post PTT

Payroll Staff
Requirements:

• Female, 23-28 years old
• Having good knowledge in Taxation (PPh 21, PPh Masa, SPT)
• Certified in Brevet Pajak A & B
• Able to operate Microsoft Windows
• Minimum 2 years experrience at Payroll Position
• Able to work in a Team

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